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Bellinger, Frederick P.

One or more keywords matched the following properties of Bellinger, Frederick P.

Property	Value
overview	Our research currently investigates the different roles of members of the selenoprotein family in brain function and neurological disorders. Selenoproteins are proteins that contain the strong antioxidant element selenium in the form of selenocysteine, 21st amino acid. We are examining the roles of these proteins in neuron physiology, synaptic plasticity, and in neurodegenerative disorders such as Alzheimer’s and Parkinson’s diseases. Our ongoing research indicates that selenoproteins may be very important for synaptic development and function, and have found correlations of specific selenoproteins with neurodegenerative disorders.

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overview

Our research currently investigates the different roles of members of the selenoprotein family in brain function and neurological disorders. Selenoproteins are proteins that contain the strong antioxidant element selenium in the form of selenocysteine, 21st amino acid. We are examining the roles of these proteins in neuron physiology, synaptic plasticity, and in neurodegenerative disorders such as Alzheimer’s and Parkinson’s diseases. Our ongoing research indicates that selenoproteins may be very important for synaptic development and function, and have found correlations of specific selenoproteins with neurodegenerative disorders.

One or more keywords matched the following items that are connected to Bellinger, Frederick P.

Item Type	Name
Academic Article	Ethanol exposure during the third trimester equivalent results in long-lasting decreased synaptic efficacy but not plasticity in the CA1 region of the rat hippocampus.
Academic Article	Regulation of presynaptic Ca2+, synaptic plasticity and contextual fear conditioning by a N-terminal ?-amyloid fragment.
Academic Article	Long-lasting synaptic modification in the rat hippocampus resulting from NMDA receptor blockade during development.
Academic Article	Developmentally regulated switch in alternatively spliced SNAP-25 isoforms alters facilitation of synaptic transmission.
Concept	Neuronal Plasticity

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Neuronal Plasticity